Multiple Sclerosis

 

By studying and analyzing the patterns of the pathocomplex process, we came to the conclusion that multiple sclerosis, parkinsonism, and Alzheimer’s disease can also be assigned to systemic group of pathologies.

The attempts of attributing the origin of these diseases to yet another virus speak of a desire to help the ill while and simultaneously demonstrate the helplessness of medicine when faced with unidentified pathologies.

Based on the theory of the pathocomplex process, both multiple sclerosis, parkinsonism and Alzheimer’s disease emerge as a result of an extensive accumulation of pathocomplexes in the vascular system of the brain. The pathocomplex occlusion of the brain vessels immediately triggers scattered infarction of its tissues with subsequent necrosis and replacement of brain tissue with fibrous niduses, triggering a manifestation of symptoms particular to one disease or another, depending on the topographic characteristics of the lesions.

In terms of physiology and biology, our brain activity consists of a series of sequential biochemical reactions that leave behind products of metabolism that must be removed with the bloodstream. Sleeping is intended exactly for this process. Every healthy human being with average level of brain activity requires an 8-hour sleep schedule for his brain to restore to a state of operationability. One can only imagine how intensive are the biochemical reactions that occur during our brain performance that even with such an extensive blood supply and such a relatively small size, it needs such a long rest for its full recovery. If our brain is in a state of hyperactivity (e.g., in stressful conditions or intensive scientific studies) and there is a simultaneous reduction in its blood supply, then the metabolites have no time to be removed and instead are deposited, eventually causing an intoxication of our brain.

Almost all cerebral diseases, except tumors, arise from a discrepancy between the rate of biochemical reactions that occur in it and the ability of the brain blood circulation to wash out metabolic products that have accumulated as a result of these reactions. As we age, due to reduction in blood circulation induced by the pathocomplex process, decomposition products fail to wash out and as a result, cerebral cells are destroyed and subsequently replaced by fibrous tissues. As noted earlier, this process is irreversible and it is the origin of multiple sclerosis, Parkinson’s and Alzheimer’s diseases.

Treatments of these pathologies based on the theory of the pathocomplex process demonstrate that symptoms of cerebral lesion become less pronounced with improved cerebral blood circulation, and in parallel, cerebral performance also increases, memory and intelligence functions recover. By eliminating the origin of systemic diseases, i.e. the pathocomplex process, the process of further blood vessel occlusion and hence, necrosis of cerebral tissues supplied by these blood vessels stops, thus suspending the spread of multiple sclerosis and progression of Parkinson’s and Alzheimer’s diseases. New niduses cease to form; however, unfortunately, previously formed niduses cannot be eliminated because they are now scars and so far, reconstructing a scar tissue back to a highly specific brain tissue is something that no one in the world can do. It is true that recently formed niduses can still recover once regenerated under the influence of the treatment process that aims to increase blood circulation. Also, once collateral circulation is improved, healthy regions of the brain can partially take over the functions of affected regions.

As a result, we can stay that a treatment aimed at addressing the source of the diseases allows to significantly reduce disease intensity and to stop further spread of multiple sclerosis, parkinsonism, and Alzheimer’s disease and for early stages, even to completely eliminate them. Of course, if extensive connective tissue has already replaced specific tissues in the brain, then in this case, even our treatment cannot yield a substantial benefit, although, as noted earlier, it can stimulate healthy regions to take over the functions of affected regions. However, at the very least, elimination of the origin of these diseases fixates the state of the disease; the disease no longer processes and no longer aggravates by the formation of new lesions and this too, is an important outcome for such patients.

 

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